By Joel David Hubbard, Joel Hubbard

ISBN-10: 0781782023

ISBN-13: 9780781782029

The second one variation bargains a concise evaluation of all components of scientific lab technological know-how, together with the normal components, equivalent to hematology, chemistry, hemostasis, immunohematology, scientific microbiology, parasitology, urinalysis and extra, in addition to lab administration, lab govt rules, and caliber coverage. A spouse site deals 35 case reports, a picture financial institution of colour pictures, and a quiz financial institution with 500 questions in certification layout.

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Extra info for A Concise Review of Clinical Laboratory Science, 2nd Edition

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The primary cause of porphyrias is a specific enzyme deficiency. a. The neurologic porphyrias include the symptoms of abdominal pain, psychotic behavior, and neuromuscular difficulties. Clinically, the three neurologic porphyrias have in common increased urinary ALA and porphobilinogen levels. (1) Acute intermittent porphyria (most common) has a clinical presentation of increased uroporphyrin levels. (2) Variegate porphyria (rare) has a presentation of increased protoporphyrin and coproporphyrin levels.

Homocystinuria is caused by impaired enzyme activity, which results in elevated levels of homocysteine and methionine in plasma and urine. 4. Protein structure. The principal plasma proteins include albumin and the globulins. Other protein fractions include fibrinogen and complement. a. Albumin is responsible for the osmotic pressure of plasma and serves as a transport protein. Prealbumin migrates ahead of albumin during electrophoresis and transports thyroid hormones. b. The globulins are insoluble in water.

Storage function. Iron, glycogen, amino acids, and some lipids are stored in hep- atocytes. 3. Hepatic disorders a. Jaundice, which causes yellowish discoloration of skin, is caused by abnormal bilirubin metabolism or by retention of bilirubin. (1) Prehepatic jaundice is the result of excessive bilirubin presented to the liver. It can occur in newborns and in people with hemolytic anemia or ineffective erythropoiesis. This condition produces increased serum unconjugated bilirubin. (2) Hepatic jaundice is present in people with hepatobiliary disease.

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A Concise Review of Clinical Laboratory Science, 2nd Edition by Joel David Hubbard, Joel Hubbard


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